Procedura valutativa per Professore di seconda fascia 2024PA533

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l documento ufficiale è reperibile all’Albo on line di Ateneo

Scadenza: 27 giugno 2024 ore 13.00

Domanda telematica

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l documento ufficiale è reperibile all’Albo on line di Ateneo

Scadenza: 27 giugno 2024 ore 13.00

Domanda telematica

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Procedura valutativa per la chiamata di un Professore di seconda fascia, ai sensi dell’art. 24, comma 5, Legge 30 dicembre 2010, n. 240, riservata a ricercatori a tempo determinato di cui all’art. 24 comma 3 lett. b) della Legge 30 dicembre 2010, n.240 nel terzo anno del contratto triennale di lavoro subordinato, a tempo determinato, stipulato con la medesima Università ed in possesso dell’Abilitazione Scientifica Nazionale ai sensi dell’art. 16 della Legge 30 dicembre 2010, n. 240 – 2024PA533 - Dipartimento di Ingegneria civile, edile e ambientale - ICEA – settore concorsuale 08/E2 – RESTAURO E STORIA DELL’ARCHITETTURA (gruppo scientifico- disciplinare 08/CEAR-11 - RESTAURO E STORIA DELL’ARCHITETTURA) – settore scientifico-disciplinare ICAR/18 – STORIA DELL’ARCHITETTURA (CEAR-11/A - STORIA DELL’ARCHITETTURA).

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Procedura valutativa per la chiamata di un Professore di seconda fascia, ai sensi dell’art. 24, comma 5, Legge 30 dicembre 2010, n. 240, riservata a ricercatori a tempo determinato di cui all’art. 24 comma 3 lett. b) della Legge 30 dicembre 2010, n.240 nel terzo anno del contratto triennale di lavoro subordinato, a tempo determinato, stipulato con la medesima Università ed in possesso dell’Abilitazione Scientifica Nazionale ai sensi dell’art. 16 della Legge 30 dicembre 2010, n. 240 – 2024PA533 - Dipartimento di Ingegneria civile, edile e ambientale - ICEA – settore concorsuale 08/E2 – RESTAURO E STORIA DELL’ARCHITETTURA (gruppo scientifico- disciplinare 08/CEAR-11 - RESTAURO E STORIA DELL’ARCHITETTURA) – settore scientifico-disciplinare ICAR/18 – STORIA DELL’ARCHITETTURA (CEAR-11/A - STORIA DELL’ARCHITETTURA).

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l documento ufficiale è reperibile all’Albo on line di Ateneo

Scadenza: 27 giugno 2024 ore 13.00

Domanda telematica

[summary] => [format] => 2 [safe_value] =>

l documento ufficiale è reperibile all’Albo on line di Ateneo

Scadenza: 27 giugno 2024 ore 13.00

Domanda telematica

[safe_summary] => ) ) [#formatter] => text_summary_or_trimmed [0] => Array ( [#markup] =>

l documento ufficiale è reperibile all’Albo on line di Ateneo

Scadenza: 27 giugno 2024 ore 13.00

Domanda telematica

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UniRe - Formazione e informazione

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Io parlo

Il progetto prevede interventi di formazione «a cascata» che, a partire dalle figure apicali dell’Ateneo (es. membri della governane, direttori e direttrici di dipartimento), sarà esteso nel corso del biennio a tutti i docenti, a studenti e studentesse e al personale tecnico amministrativo.

L’obiettivo è quello di costruire una cultura condivisa da tutta la comunità universitaria su tematiche quali stereotipi e pregiudizi di genere, linguaggio, comportamenti discriminatori, dinamiche relazionali di potere e di pericolo etc.

Sono inoltre previsti contenuti informativi per promuovere una conoscenza diffusa dei supporti che l'Università di Padova e il territorio prevedono a tutela di chi subisce molestie, violenze e discriminazioni.

 

L'Università di Padova contro la discriminazione con il progetto Università Responsabile UniRe

[summary] => [format] => filter_html_span [safe_value] =>

Io parlo

Il progetto prevede interventi di formazione «a cascata» che, a partire dalle figure apicali dell’Ateneo (es. membri della governane, direttori e direttrici di dipartimento), sarà esteso nel corso del biennio a tutti i docenti, a studenti e studentesse e al personale tecnico amministrativo.

L’obiettivo è quello di costruire una cultura condivisa da tutta la comunità universitaria su tematiche quali stereotipi e pregiudizi di genere, linguaggio, comportamenti discriminatori, dinamiche relazionali di potere e di pericolo etc.

Sono inoltre previsti contenuti informativi per promuovere una conoscenza diffusa dei supporti che l'Università di Padova e il territorio prevedono a tutela di chi subisce molestie, violenze e discriminazioni.

 

L'Università di Padova contro la discriminazione con il progetto Università Responsabile UniRe

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Ufficio Public Engagement

Area Comunicazione e Marketing

Palazzo Storione
Riviera Tito Livio 6, 35123 Padova
Tel. 049.8273537
E-mail progetto.unire@unipd.it

[format] => 2 [safe_value] =>

Ufficio Public Engagement

Area Comunicazione e Marketing

Palazzo Storione
Riviera Tito Livio 6, 35123 Padova
Tel. 049.8273537
E-mail progetto.unire@unipd.it

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Io parlo

Il progetto prevede interventi di formazione «a cascata» che, a partire dalle figure apicali dell’Ateneo (es. membri della governane, direttori e direttrici di dipartimento), sarà esteso nel corso del biennio a tutti i docenti, a studenti e studentesse e al personale tecnico amministrativo.

L’obiettivo è quello di costruire una cultura condivisa da tutta la comunità universitaria su tematiche quali stereotipi e pregiudizi di genere, linguaggio, comportamenti discriminatori, dinamiche relazionali di potere e di pericolo etc.

Sono inoltre previsti contenuti informativi per promuovere una conoscenza diffusa dei supporti che l'Università di Padova e il territorio prevedono a tutela di chi subisce molestie, violenze e discriminazioni.

 

L'Università di Padova contro la discriminazione con il progetto Università Responsabile UniRe

[summary] => [format] => filter_html_span [safe_value] =>

Io parlo

Il progetto prevede interventi di formazione «a cascata» che, a partire dalle figure apicali dell’Ateneo (es. membri della governane, direttori e direttrici di dipartimento), sarà esteso nel corso del biennio a tutti i docenti, a studenti e studentesse e al personale tecnico amministrativo.

L’obiettivo è quello di costruire una cultura condivisa da tutta la comunità universitaria su tematiche quali stereotipi e pregiudizi di genere, linguaggio, comportamenti discriminatori, dinamiche relazionali di potere e di pericolo etc.

Sono inoltre previsti contenuti informativi per promuovere una conoscenza diffusa dei supporti che l'Università di Padova e il territorio prevedono a tutela di chi subisce molestie, violenze e discriminazioni.

 

L'Università di Padova contro la discriminazione con il progetto Università Responsabile UniRe

[safe_summary] => ) ) [#formatter] => text_summary_or_trimmed [0] => Array ( [#markup] =>

Io parlo

Il progetto prevede interventi di formazione «a cascata» che, a partire dalle figure apicali dell’Ateneo (es. membri della governane, direttori e direttrici di dipartimento), sarà esteso nel corso del biennio a tutti i docenti, a studenti e studentesse e al personale tecnico amministrativo.

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Io parlo

Il progetto prevede interventi di formazione «a cascata» che, a partire dalle figure apicali dell’Ateneo (es. membri della governane, direttori e direttrici di dipartimento), sarà esteso nel corso del biennio a tutti i docenti, a studenti e studentesse e al personale tecnico amministrativo.

L’obiettivo è quello di costruire una cultura condivisa da tutta la comunità universitaria su tematiche quali stereotipi e pregiudizi di genere, linguaggio, comportamenti discriminatori, dinamiche relazionali di potere e di pericolo etc.

Sono inoltre previsti contenuti informativi per promuovere una conoscenza diffusa dei supporti che l'Università di Padova e il territorio prevedono a tutela di chi subisce molestie, violenze e discriminazioni.

 

L'Università di Padova contro la discriminazione con il progetto Università Responsabile UniRe

[summary] => [format] => filter_html_span [safe_value] =>

Io parlo

Il progetto prevede interventi di formazione «a cascata» che, a partire dalle figure apicali dell’Ateneo (es. membri della governane, direttori e direttrici di dipartimento), sarà esteso nel corso del biennio a tutti i docenti, a studenti e studentesse e al personale tecnico amministrativo.

L’obiettivo è quello di costruire una cultura condivisa da tutta la comunità universitaria su tematiche quali stereotipi e pregiudizi di genere, linguaggio, comportamenti discriminatori, dinamiche relazionali di potere e di pericolo etc.

Sono inoltre previsti contenuti informativi per promuovere una conoscenza diffusa dei supporti che l'Università di Padova e il territorio prevedono a tutela di chi subisce molestie, violenze e discriminazioni.

 

L'Università di Padova contro la discriminazione con il progetto Università Responsabile UniRe

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Ufficio Public Engagement

Area Comunicazione e Marketing

Palazzo Storione
Riviera Tito Livio 6, 35123 Padova
Tel. 049.8273537
E-mail progetto.unire@unipd.it

[format] => 2 [safe_value] =>

Ufficio Public Engagement

Area Comunicazione e Marketing

Palazzo Storione
Riviera Tito Livio 6, 35123 Padova
Tel. 049.8273537
E-mail progetto.unire@unipd.it

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Ufficio Public Engagement

Area Comunicazione e Marketing

Palazzo Storione
Riviera Tito Livio 6, 35123 Padova
Tel. 049.8273537
E-mail progetto.unire@unipd.it

[format] => 2 [safe_value] =>

Ufficio Public Engagement

Area Comunicazione e Marketing

Palazzo Storione
Riviera Tito Livio 6, 35123 Padova
Tel. 049.8273537
E-mail progetto.unire@unipd.it

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Ufficio Public Engagement

Area Comunicazione e Marketing

Palazzo Storione
Riviera Tito Livio 6, 35123 Padova
Tel. 049.8273537
E-mail progetto.unire@unipd.it

[format] => 2 [safe_value] =>

Ufficio Public Engagement

Area Comunicazione e Marketing

Palazzo Storione
Riviera Tito Livio 6, 35123 Padova
Tel. 049.8273537
E-mail progetto.unire@unipd.it

) ) ) [name] => stefano.zampieri [picture] => 0 [data] => a:2:{s:13:"form_build_id";s:48:"form-WsCySmos4vAVlyFhG6gU5T7knfAyqco8LxlocSU_yIA";s:14:"wysiwyg_status";a:1:{i:1;i:1;}} [num_revisions] => 2 [current_revision_id] => 494947 [is_current] => 1 [is_pending] => [revision_moderation] => ) ) ) ) )

Unipd Research - Unveiling a New Gene that Regulates Aging

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

The identification of this new gene is thanks to the collaboration with Eva Trevisson, geneticist from the Department of Women's and Children's Health at the University of Padua, and nine years of work between Italian and internationally renowned researchers. Published in the prestigious medical journal of translational research The Journal of Clinical Investigation the work entitled C16ORF70/Mytho promotes healthy ageing in C. elegans and prevents cellular senescence in mammalsreceived funding from a PNRR action on aging. The action, called AGE-IT Ageing Well in an Aging Society aims to create a national network of researchers studying this biological process.

Main co-author, together with Dr. Valeria Morbidoni, Dr. Anais Franco Romero explains 'The work began with computer research to identify potential, yet unknown, genes in the human genome that could be relevant to the mechanisms that control the quality of proteins and organelles. Among the various candidates, the team focused on a gene that stood out for being extremely conserved among different animal species, from humans to worms, called the MYTHO protein (Macroautophagy and YouTH Optimizer)'.

Through genetic manipulation experiments, the group demonstrated that the inhibition of this protein causes early cellular senescence when cells stop replicating, thus shortening of lifespan of the animal model Caenorhabditis Elegans, while its activation improves the quality of life and maintains healthy aging.

Also characterizing the molecular mechanisms, the study also discovered that this gene regulates autophagy, which removes damaged proteins and organelles, improving cellular homeostasis.

Prof Marco Sandri explains “Even after years of studies, we have come to know something about our genome, namely that the function of most of our genetic code is still unknown. An example is the genes that encode proteins, of which more than 5000 out of a total of 20000 are completely unknown. For this reason, in recent years we have used resources and energy to characterize this unknown world of our DNA.”

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

The identification of this new gene is thanks to the collaboration with Eva Trevisson, geneticist from the Department of Women's and Children's Health at the University of Padua, and nine years of work between Italian and internationally renowned researchers. Published in the prestigious medical journal of translational research The Journal of Clinical Investigation the work entitled C16ORF70/Mytho promotes healthy ageing in C. elegans and prevents cellular senescence in mammalsreceived funding from a PNRR action on aging. The action, called AGE-IT Ageing Well in an Aging Society aims to create a national network of researchers studying this biological process.

Main co-author, together with Dr. Valeria Morbidoni, Dr. Anais Franco Romero explains 'The work began with computer research to identify potential, yet unknown, genes in the human genome that could be relevant to the mechanisms that control the quality of proteins and organelles. Among the various candidates, the team focused on a gene that stood out for being extremely conserved among different animal species, from humans to worms, called the MYTHO protein (Macroautophagy and YouTH Optimizer)'.

Through genetic manipulation experiments, the group demonstrated that the inhibition of this protein causes early cellular senescence when cells stop replicating, thus shortening of lifespan of the animal model Caenorhabditis Elegans, while its activation improves the quality of life and maintains healthy aging.

Also characterizing the molecular mechanisms, the study also discovered that this gene regulates autophagy, which removes damaged proteins and organelles, improving cellular homeostasis.

Prof Marco Sandri explains “Even after years of studies, we have come to know something about our genome, namely that the function of most of our genetic code is still unknown. An example is the genes that encode proteins, of which more than 5000 out of a total of 20000 are completely unknown. For this reason, in recent years we have used resources and energy to characterize this unknown world of our DNA.”

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

The identification of this new gene is thanks to the collaboration with Eva Trevisson, geneticist from the Department of Women's and Children's Health at the University of Padua, and nine years of work between Italian and internationally renowned researchers. Published in the prestigious medical journal of translational research The Journal of Clinical Investigation the work entitled C16ORF70/Mytho promotes healthy ageing in C. elegans and prevents cellular senescence in mammalsreceived funding from a PNRR action on aging. The action, called AGE-IT Ageing Well in an Aging Society aims to create a national network of researchers studying this biological process.

Main co-author, together with Dr. Valeria Morbidoni, Dr. Anais Franco Romero explains 'The work began with computer research to identify potential, yet unknown, genes in the human genome that could be relevant to the mechanisms that control the quality of proteins and organelles. Among the various candidates, the team focused on a gene that stood out for being extremely conserved among different animal species, from humans to worms, called the MYTHO protein (Macroautophagy and YouTH Optimizer)'.

Through genetic manipulation experiments, the group demonstrated that the inhibition of this protein causes early cellular senescence when cells stop replicating, thus shortening of lifespan of the animal model Caenorhabditis Elegans, while its activation improves the quality of life and maintains healthy aging.

Also characterizing the molecular mechanisms, the study also discovered that this gene regulates autophagy, which removes damaged proteins and organelles, improving cellular homeostasis.

Prof Marco Sandri explains “Even after years of studies, we have come to know something about our genome, namely that the function of most of our genetic code is still unknown. An example is the genes that encode proteins, of which more than 5000 out of a total of 20000 are completely unknown. For this reason, in recent years we have used resources and energy to characterize this unknown world of our DNA.”

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

The identification of this new gene is thanks to the collaboration with Eva Trevisson, geneticist from the Department of Women's and Children's Health at the University of Padua, and nine years of work between Italian and internationally renowned researchers. Published in the prestigious medical journal of translational research The Journal of Clinical Investigation the work entitled C16ORF70/Mytho promotes healthy ageing in C. elegans and prevents cellular senescence in mammalsreceived funding from a PNRR action on aging. The action, called AGE-IT Ageing Well in an Aging Society aims to create a national network of researchers studying this biological process.

Main co-author, together with Dr. Valeria Morbidoni, Dr. Anais Franco Romero explains 'The work began with computer research to identify potential, yet unknown, genes in the human genome that could be relevant to the mechanisms that control the quality of proteins and organelles. Among the various candidates, the team focused on a gene that stood out for being extremely conserved among different animal species, from humans to worms, called the MYTHO protein (Macroautophagy and YouTH Optimizer)'.

Through genetic manipulation experiments, the group demonstrated that the inhibition of this protein causes early cellular senescence when cells stop replicating, thus shortening of lifespan of the animal model Caenorhabditis Elegans, while its activation improves the quality of life and maintains healthy aging.

Also characterizing the molecular mechanisms, the study also discovered that this gene regulates autophagy, which removes damaged proteins and organelles, improving cellular homeostasis.

Prof Marco Sandri explains “Even after years of studies, we have come to know something about our genome, namely that the function of most of our genetic code is still unknown. An example is the genes that encode proteins, of which more than 5000 out of a total of 20000 are completely unknown. For this reason, in recent years we have used resources and energy to characterize this unknown world of our DNA.”

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

The identification of this new gene is thanks to the collaboration with Eva Trevisson, geneticist from the Department of Women's and Children's Health at the University of Padua, and nine years of work between Italian and internationally renowned researchers. Published in the prestigious medical journal of translational research The Journal of Clinical Investigation the work entitled C16ORF70/Mytho promotes healthy ageing in C. elegans and prevents cellular senescence in mammalsreceived funding from a PNRR action on aging. The action, called AGE-IT Ageing Well in an Aging Society aims to create a national network of researchers studying this biological process.

Main co-author, together with Dr. Valeria Morbidoni, Dr. Anais Franco Romero explains 'The work began with computer research to identify potential, yet unknown, genes in the human genome that could be relevant to the mechanisms that control the quality of proteins and organelles. Among the various candidates, the team focused on a gene that stood out for being extremely conserved among different animal species, from humans to worms, called the MYTHO protein (Macroautophagy and YouTH Optimizer)'.

Through genetic manipulation experiments, the group demonstrated that the inhibition of this protein causes early cellular senescence when cells stop replicating, thus shortening of lifespan of the animal model Caenorhabditis Elegans, while its activation improves the quality of life and maintains healthy aging.

Also characterizing the molecular mechanisms, the study also discovered that this gene regulates autophagy, which removes damaged proteins and organelles, improving cellular homeostasis.

Prof Marco Sandri explains “Even after years of studies, we have come to know something about our genome, namely that the function of most of our genetic code is still unknown. An example is the genes that encode proteins, of which more than 5000 out of a total of 20000 are completely unknown. For this reason, in recent years we have used resources and energy to characterize this unknown world of our DNA.”

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

The identification of this new gene is thanks to the collaboration with Eva Trevisson, geneticist from the Department of Women's and Children's Health at the University of Padua, and nine years of work between Italian and internationally renowned researchers. Published in the prestigious medical journal of translational research The Journal of Clinical Investigation the work entitled C16ORF70/Mytho promotes healthy ageing in C. elegans and prevents cellular senescence in mammalsreceived funding from a PNRR action on aging. The action, called AGE-IT Ageing Well in an Aging Society aims to create a national network of researchers studying this biological process.

Main co-author, together with Dr. Valeria Morbidoni, Dr. Anais Franco Romero explains 'The work began with computer research to identify potential, yet unknown, genes in the human genome that could be relevant to the mechanisms that control the quality of proteins and organelles. Among the various candidates, the team focused on a gene that stood out for being extremely conserved among different animal species, from humans to worms, called the MYTHO protein (Macroautophagy and YouTH Optimizer)'.

Through genetic manipulation experiments, the group demonstrated that the inhibition of this protein causes early cellular senescence when cells stop replicating, thus shortening of lifespan of the animal model Caenorhabditis Elegans, while its activation improves the quality of life and maintains healthy aging.

Also characterizing the molecular mechanisms, the study also discovered that this gene regulates autophagy, which removes damaged proteins and organelles, improving cellular homeostasis.

Prof Marco Sandri explains “Even after years of studies, we have come to know something about our genome, namely that the function of most of our genetic code is still unknown. An example is the genes that encode proteins, of which more than 5000 out of a total of 20000 are completely unknown. For this reason, in recent years we have used resources and energy to characterize this unknown world of our DNA.”

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

The identification of this new gene is thanks to the collaboration with Eva Trevisson, geneticist from the Department of Women's and Children's Health at the University of Padua, and nine years of work between Italian and internationally renowned researchers. Published in the prestigious medical journal of translational research The Journal of Clinical Investigation the work entitled C16ORF70/Mytho promotes healthy ageing in C. elegans and prevents cellular senescence in mammalsreceived funding from a PNRR action on aging. The action, called AGE-IT Ageing Well in an Aging Society aims to create a national network of researchers studying this biological process.

Main co-author, together with Dr. Valeria Morbidoni, Dr. Anais Franco Romero explains 'The work began with computer research to identify potential, yet unknown, genes in the human genome that could be relevant to the mechanisms that control the quality of proteins and organelles. Among the various candidates, the team focused on a gene that stood out for being extremely conserved among different animal species, from humans to worms, called the MYTHO protein (Macroautophagy and YouTH Optimizer)'.

Through genetic manipulation experiments, the group demonstrated that the inhibition of this protein causes early cellular senescence when cells stop replicating, thus shortening of lifespan of the animal model Caenorhabditis Elegans, while its activation improves the quality of life and maintains healthy aging.

Also characterizing the molecular mechanisms, the study also discovered that this gene regulates autophagy, which removes damaged proteins and organelles, improving cellular homeostasis.

Prof Marco Sandri explains “Even after years of studies, we have come to know something about our genome, namely that the function of most of our genetic code is still unknown. An example is the genes that encode proteins, of which more than 5000 out of a total of 20000 are completely unknown. For this reason, in recent years we have used resources and energy to characterize this unknown world of our DNA.”

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

The identification of this new gene is thanks to the collaboration with Eva Trevisson, geneticist from the Department of Women's and Children's Health at the University of Padua, and nine years of work between Italian and internationally renowned researchers. Published in the prestigious medical journal of translational research The Journal of Clinical Investigation the work entitled C16ORF70/Mytho promotes healthy ageing in C. elegans and prevents cellular senescence in mammalsreceived funding from a PNRR action on aging. The action, called AGE-IT Ageing Well in an Aging Society aims to create a national network of researchers studying this biological process.

Main co-author, together with Dr. Valeria Morbidoni, Dr. Anais Franco Romero explains 'The work began with computer research to identify potential, yet unknown, genes in the human genome that could be relevant to the mechanisms that control the quality of proteins and organelles. Among the various candidates, the team focused on a gene that stood out for being extremely conserved among different animal species, from humans to worms, called the MYTHO protein (Macroautophagy and YouTH Optimizer)'.

Through genetic manipulation experiments, the group demonstrated that the inhibition of this protein causes early cellular senescence when cells stop replicating, thus shortening of lifespan of the animal model Caenorhabditis Elegans, while its activation improves the quality of life and maintains healthy aging.

Also characterizing the molecular mechanisms, the study also discovered that this gene regulates autophagy, which removes damaged proteins and organelles, improving cellular homeostasis.

Prof Marco Sandri explains “Even after years of studies, we have come to know something about our genome, namely that the function of most of our genetic code is still unknown. An example is the genes that encode proteins, of which more than 5000 out of a total of 20000 are completely unknown. For this reason, in recent years we have used resources and energy to characterize this unknown world of our DNA.”

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

The identification of this new gene is thanks to the collaboration with Eva Trevisson, geneticist from the Department of Women's and Children's Health at the University of Padua, and nine years of work between Italian and internationally renowned researchers. Published in the prestigious medical journal of translational research The Journal of Clinical Investigation the work entitled C16ORF70/Mytho promotes healthy ageing in C. elegans and prevents cellular senescence in mammalsreceived funding from a PNRR action on aging. The action, called AGE-IT Ageing Well in an Aging Society aims to create a national network of researchers studying this biological process.

Main co-author, together with Dr. Valeria Morbidoni, Dr. Anais Franco Romero explains 'The work began with computer research to identify potential, yet unknown, genes in the human genome that could be relevant to the mechanisms that control the quality of proteins and organelles. Among the various candidates, the team focused on a gene that stood out for being extremely conserved among different animal species, from humans to worms, called the MYTHO protein (Macroautophagy and YouTH Optimizer)'.

Through genetic manipulation experiments, the group demonstrated that the inhibition of this protein causes early cellular senescence when cells stop replicating, thus shortening of lifespan of the animal model Caenorhabditis Elegans, while its activation improves the quality of life and maintains healthy aging.

Also characterizing the molecular mechanisms, the study also discovered that this gene regulates autophagy, which removes damaged proteins and organelles, improving cellular homeostasis.

Prof Marco Sandri explains “Even after years of studies, we have come to know something about our genome, namely that the function of most of our genetic code is still unknown. An example is the genes that encode proteins, of which more than 5000 out of a total of 20000 are completely unknown. For this reason, in recent years we have used resources and energy to characterize this unknown world of our DNA.”

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Directed by Prof Marco Sandri of the Department of Biomedical Sciences at the University of Padua and Principal Investigator of the Veneto Institute of Molecular Medicine (VIMM), an international research team identifies and characterizes a new gene that controls cellular aging and longevity.

The identification of this new gene is thanks to the collaboration with Eva Trevisson, geneticist from the Department of Women's and Children's Health at the University of Padua, and nine years of work between Italian and internationally renowned researchers. Published in the prestigious medical journal of translational research The Journal of Clinical Investigation the work entitled C16ORF70/Mytho promotes healthy ageing in C. elegans and prevents cellular senescence in mammalsreceived funding from a PNRR action on aging. The action, called AGE-IT Ageing Well in an Aging Society aims to create a national network of researchers studying this biological process.

Main co-author, together with Dr. Valeria Morbidoni, Dr. Anais Franco Romero explains 'The work began with computer research to identify potential, yet unknown, genes in the human genome that could be relevant to the mechanisms that control the quality of proteins and organelles. Among the various candidates, the team focused on a gene that stood out for being extremely conserved among different animal species, from humans to worms, called the MYTHO protein (Macroautophagy and YouTH Optimizer)'.

Through genetic manipulation experiments, the group demonstrated that the inhibition of this protein causes early cellular senescence when cells stop replicating, thus shortening of lifespan of the animal model Caenorhabditis Elegans, while its activation improves the quality of life and maintains healthy aging.

Also characterizing the molecular mechanisms, the study also discovered that this gene regulates autophagy, which removes damaged proteins and organelles, improving cellular homeostasis.

Prof Marco Sandri explains “Even after years of studies, we have come to know something about our genome, namely that the function of most of our genetic code is still unknown. An example is the genes that encode proteins, of which more than 5000 out of a total of 20000 are completely unknown. For this reason, in recent years we have used resources and energy to characterize this unknown world of our DNA.”

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Risultati CdA 25 giugno 2024

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2024S41 Calendario e sedi prove d'esame

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An Innovative Approach to Treat Brain Tumors

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Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

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Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

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Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

[summary] => [format] => 2 [safe_value] =>

Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

[safe_summary] => ) ) ) [field_date_box_lancio_news] => Array ( [und] => Array ( [0] => Array ( [value] => 2024-06-25T00:00:00 [timezone] => Europe/Paris [timezone_db] => Europe/Paris [date_type] => date ) ) ) [field_etichetta_box_lancio_news] => Array ( ) [field_img_box_lancio_news] => Array ( [und] => Array ( [0] => Array ( [fid] => 131630 [uid] => 2032 [filename] => n_cervelli_5.jpg [uri] => public://n_cervelli_5_0.jpg [filemime] => image/jpeg [filesize] => 33627 [status] => 1 [timestamp] => 1719311452 [type] => image [field_file_image_alt_text] => Array ( ) [field_file_image_title_text] => Array ( ) [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2048 ) ) ) [metadata] => Array ( [height] => 227 [width] => 677 ) [height] => 227 [width] => 677 [alt] => brains [title] => ) ) ) [field_link_alla_news] => Array ( ) [field_link_esterno_news] => Array ( [und] => Array ( [0] => Array ( [value] => [format] => [safe_value] => ) ) ) [field_pagina_associata] => Array ( ) [field_link_etichetta] => Array ( ) [field_abstract_news] => Array ( [und] => Array ( [0] => Array ( [value] => Multidisciplinary Padua team discovers role of brain connections in tumor growth [format] => [safe_value] => Multidisciplinary Padua team discovers role of brain connections in tumor growth ) ) ) [field_allegato_news] => Array ( ) [field_categorie_news] => Array ( [und] => Array ( [0] => Array ( [tid] => 2296 ) ) ) [field_pub_date] => Array ( [und] => Array ( [0] => Array ( [value] => 2024-06-25T00:00:00 [value2] => 2025-06-25T00:00:00 [timezone] => Europe/Paris [timezone_db] => Europe/Paris [date_type] => date ) ) ) [field_layout_news] => Array ( [und] => Array ( [0] => Array ( [value] => single ) ) ) [field_testo_opzionale_news] => Array ( ) [field_url_en_page] => Array ( ) [field_url_en_page_label] => Array ( ) [path] => Array ( [pathauto] => 1 ) [name] => francesca.forzan [picture] => 0 [data] => b:0; [num_revisions] => 1 [current_revision_id] => 462336 [is_current] => 1 [is_pending] => [revision_moderation] => [entity_view_prepared] => 1 ) [#items] => Array ( [0] => Array ( [value] =>

Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

[summary] => [format] => 2 [safe_value] =>

Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

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Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

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Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

[summary] => [format] => 2 [safe_value] =>

Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

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Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

[summary] => [format] => 2 [safe_value] =>

Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

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Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

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Despite substantial research efforts over the past 30 years, the prognosis for those suffering from brain tumors remains bleak, why is that?

Directed by Prof Maurizio Corbetta (Department of Neuroscience and VIMM), the work of the multidisciplinary research team from the University of Padua, which includes Alessandro Salvalaggio (neurologist), Lorenzo Pini (psychologist) and Alessandra Bertoldo (engineer), aims to answer this.

Limited advancements in treatment are due to the idea that glioblastoma (brain tumors) is like any other type of tumor of any other organ. Rather, Unipd researchers consider that brain tumor growth is partly determined and regulated by brain activities within itself.

The brain is the most complex organ in the human body, holding approximately 100 billion neurons. These neurons organize among themselves according to a complex structure (structural connectome) and form specific activity networks (functional connectome). Tumor cells integrate into the connectome by exploiting its structural and functional connections toward growth and metastasis. Consequently, the study of brain connectivity takes on a new role in determining the prognosis of these patients, but above all, providing new treatment strategies.

Studies include those recently published in Jama Neurology, where authors show that a patient's survival can be predicted under a newly developed white matter tract density index (TDI), whereas other possible future applications involve radiotherapy and surgical navigation.

Furthermore, modulating connectivity could slow down tumor growth by providing patients with new treatments that complement traditional chemotherapy and radiotherapy interventions, says Maurizio Corbetta.

The study Glioblastoma and brain connectivity: the need for a paradigm shift was published in Lancet Neurology, the world’s most important neurology journal.

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Graduatoria Scuola Primaria - prove orali - Sostegno 2023-24

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DALLE ZONE A RISCHIO ALL’UNIVERSITÀ DI PADOVA: IL FUNDRAISING PER LE OPPORTUNITÀ DI STUDIO. Giovedì 27 giugno l’incontro in Aula Nievo di Palazzo Bo

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Giovedì 27 giugno l’incontro in Aula Nievo di Palazzo Bo [log] => [status] => 1 [comment] => 0 [promote] => 1 [sticky] => 0 [nid] => 111601 [type] => allegato_area_stampa [language] => und [created] => 1719307705 [changed] => 1719307705 [tnid] => 0 [translate] => 0 [revision_timestamp] => 1719307705 [revision_uid] => 8835 [body] => Array ( ) [field_allegato_area_stampa] => Array ( [und] => Array ( [0] => Array ( [fid] => 131622 [uid] => 8835 [filename] => 2024-06-25_Students at risk_incontro 27 giugno.pdf [uri] => public://2024-06-25_Students at risk_incontro 27 giugno.pdf [filemime] => application/pdf [filesize] => 380441 [status] => 1 [timestamp] => 1719307705 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2048 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) ) [field_all_imm_area_stampa] => Array ( ) [field_data_area_stampa] => Array ( [und] => Array ( [0] => Array ( [value] => 2024-06-25 00:00:00 [timezone] => Europe/Paris [timezone_db] => Europe/Paris [date_type] => datetime ) ) ) [field_luogo_area_stampa] => Array ( [und] => Array ( [0] => Array ( [value] => Padova [format] => [safe_value] => Padova ) ) ) [name] => stampa [picture] => 0 [data] => b:0; [num_revisions] => 1 [current_revision_id] => 462313 [is_current] => 1 [is_pending] => [revision_moderation] => [entity_view_prepared] => 1 ) [#items] => Array ( [0] => Array ( [value] => 2024-06-25 00:00:00 [timezone] => Europe/Paris [timezone_db] => Europe/Paris [date_type] => datetime ) ) [#formatter] => date_default [0] => Array ( [#markup] => Mar, 25/06/2024 ) ) [field_allegato_area_stampa] => Array ( [#theme] => field [#weight] => -2 [#title] => Allegato [#access] => 1 [#label_display] => above [#view_mode] => teaser [#language] => und [#field_name] => field_allegato_area_stampa [#field_type] => file [#field_translatable] => 0 [#entity_type] => node [#bundle] => allegato_area_stampa [#object] => stdClass Object ( [vid] => 462313 [uid] => 8835 [title] => DALLE ZONE A RISCHIO ALL’UNIVERSITÀ DI PADOVA: IL FUNDRAISING PER LE OPPORTUNITÀ DI STUDIO. Giovedì 27 giugno l’incontro in Aula Nievo di Palazzo Bo [log] => [status] => 1 [comment] => 0 [promote] => 1 [sticky] => 0 [nid] => 111601 [type] => allegato_area_stampa [language] => und [created] => 1719307705 [changed] => 1719307705 [tnid] => 0 [translate] => 0 [revision_timestamp] => 1719307705 [revision_uid] => 8835 [body] => Array ( ) [field_allegato_area_stampa] => Array ( [und] => Array ( [0] => Array ( [fid] => 131622 [uid] => 8835 [filename] => 2024-06-25_Students at risk_incontro 27 giugno.pdf [uri] => public://2024-06-25_Students at risk_incontro 27 giugno.pdf [filemime] => application/pdf [filesize] => 380441 [status] => 1 [timestamp] => 1719307705 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2048 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) ) [field_all_imm_area_stampa] => Array ( ) [field_data_area_stampa] => Array ( [und] => Array ( [0] => Array ( [value] => 2024-06-25 00:00:00 [timezone] => Europe/Paris [timezone_db] => Europe/Paris [date_type] => datetime ) ) ) [field_luogo_area_stampa] => Array ( [und] => Array ( [0] => Array ( [value] => Padova [format] => [safe_value] => Padova ) ) ) [name] => stampa [picture] => 0 [data] => b:0; [num_revisions] => 1 [current_revision_id] => 462313 [is_current] => 1 [is_pending] => [revision_moderation] => [entity_view_prepared] => 1 ) [#items] => Array ( [0] => Array ( [fid] => 131622 [uid] => 8835 [filename] => 2024-06-25_Students at risk_incontro 27 giugno.pdf [uri] => public://2024-06-25_Students at risk_incontro 27 giugno.pdf [filemime] => application/pdf [filesize] => 380441 [status] => 1 [timestamp] => 1719307705 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2048 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) [#formatter] => file_default [0] => Array ( [#theme] => file_link [#file] => stdClass Object ( [fid] => 131622 [uid] => 8835 [filename] => 2024-06-25_Students at risk_incontro 27 giugno.pdf [uri] => public://2024-06-25_Students at risk_incontro 27 giugno.pdf [filemime] => application/pdf [filesize] => 380441 [status] => 1 [timestamp] => 1719307705 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2048 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) ) [links] => Array ( [#theme] => links__node [#pre_render] => Array ( [0] => drupal_pre_render_links ) [#attributes] => Array ( [class] => Array ( [0] => links [1] => inline ) ) [node] => Array ( [#theme] => links__node__node [#links] => Array ( [node-readmore] => Array ( [title] => Read more about DALLE ZONE A RISCHIO ALL’UNIVERSITÀ DI PADOVA: IL FUNDRAISING PER LE OPPORTUNITÀ DI STUDIO. Giovedì 27 giugno l’incontro in Aula Nievo di Palazzo Bo [href] => node/111601 [html] => 1 [attributes] => Array ( [rel] => tag [title] => DALLE ZONE A RISCHIO ALL’UNIVERSITÀ DI PADOVA: IL FUNDRAISING PER LE OPPORTUNITÀ DI STUDIO. Giovedì 27 giugno l’incontro in Aula Nievo di Palazzo Bo ) ) ) [#attributes] => Array ( [class] => Array ( [0] => links [1] => inline ) ) ) ) )

2024RUA03 - Allegato 2 - Verbale 1 - Criteri

Array ( [field_titolo_frontend_all] => Array ( [#theme] => field [#weight] => -4 [#title] => Titolo frontend [#access] => 1 [#label_display] => above [#view_mode] => teaser [#language] => und [#field_name] => field_titolo_frontend_all [#field_type] => text_long [#field_translatable] => 0 [#entity_type] => node [#bundle] => allegato [#object] => stdClass Object ( [vid] => 462311 [uid] => 8831 [title] => 2024RUA03 - Allegato 2 - Verbale 1 - Criteri [log] => [status] => 1 [comment] => 0 [promote] => 1 [sticky] => 0 [nid] => 111600 [type] => allegato [language] => it [created] => 1719307276 [changed] => 1720083756 [tnid] => 0 [translate] => 0 [revision_timestamp] => 1720083756 [revision_uid] => 102 [taxonomy_vocabulary_2] => Array ( ) [taxonomy_vocabulary_8] => Array ( ) [body] => Array ( ) [field_titolo_frontend_all] => Array ( [und] => Array ( [0] => Array ( [value] => Verbale 1 - Criteri [format] => [safe_value] => Verbale 1 - Criteri ) ) ) [field_allegato_file] => Array ( [und] => Array ( [0] => Array ( [fid] => 131621 [uid] => 32 [filename] => VERBALE 1 RTDA_ ICAR 04.pdf.pdf [uri] => public://2024/VERBALE 1 RTDA_ ICAR 04.pdf.pdf [filemime] => application/pdf [filesize] => 146258 [status] => 1 [timestamp] => 1719307266 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2614 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) ) [name] => carriere.docenti [picture] => 0 [data] => b:0; [num_revisions] => 1 [current_revision_id] => 462311 [is_current] => 1 [is_pending] => [revision_moderation] => [entity_view_prepared] => 1 ) [#items] => Array ( [0] => Array ( [value] => Verbale 1 - Criteri [format] => [safe_value] => Verbale 1 - Criteri ) ) [#formatter] => text_default [0] => Array ( [#markup] => Verbale 1 - Criteri ) ) [field_allegato_file] => Array ( [#theme] => field [#weight] => -3 [#title] => File [#access] => 1 [#label_display] => above [#view_mode] => teaser [#language] => und [#field_name] => field_allegato_file [#field_type] => file [#field_translatable] => 0 [#entity_type] => node [#bundle] => allegato [#object] => stdClass Object ( [vid] => 462311 [uid] => 8831 [title] => 2024RUA03 - Allegato 2 - Verbale 1 - Criteri [log] => [status] => 1 [comment] => 0 [promote] => 1 [sticky] => 0 [nid] => 111600 [type] => allegato [language] => it [created] => 1719307276 [changed] => 1720083756 [tnid] => 0 [translate] => 0 [revision_timestamp] => 1720083756 [revision_uid] => 102 [taxonomy_vocabulary_2] => Array ( ) [taxonomy_vocabulary_8] => Array ( ) [body] => Array ( ) [field_titolo_frontend_all] => Array ( [und] => Array ( [0] => Array ( [value] => Verbale 1 - Criteri [format] => [safe_value] => Verbale 1 - Criteri ) ) ) [field_allegato_file] => Array ( [und] => Array ( [0] => Array ( [fid] => 131621 [uid] => 32 [filename] => VERBALE 1 RTDA_ ICAR 04.pdf.pdf [uri] => public://2024/VERBALE 1 RTDA_ ICAR 04.pdf.pdf [filemime] => application/pdf [filesize] => 146258 [status] => 1 [timestamp] => 1719307266 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2614 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) ) [name] => carriere.docenti [picture] => 0 [data] => b:0; [num_revisions] => 1 [current_revision_id] => 462311 [is_current] => 1 [is_pending] => [revision_moderation] => [entity_view_prepared] => 1 ) [#items] => Array ( [0] => Array ( [fid] => 131621 [uid] => 32 [filename] => VERBALE 1 RTDA_ ICAR 04.pdf.pdf [uri] => public://2024/VERBALE 1 RTDA_ ICAR 04.pdf.pdf [filemime] => application/pdf [filesize] => 146258 [status] => 1 [timestamp] => 1719307266 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2614 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) [#formatter] => file_default [0] => Array ( [#theme] => file_link [#file] => stdClass Object ( [fid] => 131621 [uid] => 32 [filename] => VERBALE 1 RTDA_ ICAR 04.pdf.pdf [uri] => public://2024/VERBALE 1 RTDA_ ICAR 04.pdf.pdf [filemime] => application/pdf [filesize] => 146258 [status] => 1 [timestamp] => 1719307266 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2614 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) ) [links] => Array ( [#theme] => links__node [#pre_render] => Array ( [0] => drupal_pre_render_links ) [#attributes] => Array ( [class] => Array ( [0] => links [1] => inline ) ) [node] => Array ( [#theme] => links__node__node [#links] => Array ( [node-readmore] => Array ( [title] => Read more about 2024RUA03 - Allegato 2 - Verbale 1 - Criteri [href] => node/111600 [html] => 1 [attributes] => Array ( [rel] => tag [title] => 2024RUA03 - Allegato 2 - Verbale 1 - Criteri ) ) ) [#attributes] => Array ( [class] => Array ( [0] => links [1] => inline ) ) ) ) )

2024RTT02_RISERVATO - Allegato 11 - Verbale 2 - Elenco candidati e convocazione

Array ( [field_titolo_frontend_all] => Array ( [#theme] => field [#weight] => -4 [#title] => Titolo frontend [#access] => 1 [#label_display] => above [#view_mode] => teaser [#language] => und [#field_name] => field_titolo_frontend_all [#field_type] => text_long [#field_translatable] => 0 [#entity_type] => node [#bundle] => allegato [#object] => stdClass Object ( [vid] => 462310 [uid] => 8831 [title] => 2024RTT02_RISERVATO - Allegato 11 - Verbale 2 - Elenco candidati e convocazione [log] => [status] => 1 [comment] => 0 [promote] => 1 [sticky] => 0 [nid] => 111599 [type] => allegato [language] => it [created] => 1719304975 [changed] => 1720083926 [tnid] => 0 [translate] => 0 [revision_timestamp] => 1720083926 [revision_uid] => 102 [taxonomy_vocabulary_2] => Array ( ) [taxonomy_vocabulary_8] => Array ( ) [body] => Array ( ) [field_titolo_frontend_all] => Array ( [und] => Array ( [0] => Array ( [value] => Verbale 2 - Elenco candidati e convocazione [format] => [safe_value] => Verbale 2 - Elenco candidati e convocazione ) ) ) [field_allegato_file] => Array ( [und] => Array ( [0] => Array ( [fid] => 131620 [uid] => 32 [filename] => Verbale 2 ELENCO E CONVOCAZIONE - 2024RTT02 RISERVATO ALL.11 IUS13.pdf.pdf [uri] => public://2024/Verbale 2 ELENCO E CONVOCAZIONE - 2024RTT02 RISERVATO ALL.11 IUS13.pdf.pdf [filemime] => application/pdf [filesize] => 310710 [status] => 1 [timestamp] => 1719304966 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2614 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) ) [name] => carriere.docenti [picture] => 0 [data] => b:0; [num_revisions] => 1 [current_revision_id] => 462310 [is_current] => 1 [is_pending] => [revision_moderation] => [entity_view_prepared] => 1 ) [#items] => Array ( [0] => Array ( [value] => Verbale 2 - Elenco candidati e convocazione [format] => [safe_value] => Verbale 2 - Elenco candidati e convocazione ) ) [#formatter] => text_default [0] => Array ( [#markup] => Verbale 2 - Elenco candidati e convocazione ) ) [field_allegato_file] => Array ( [#theme] => field [#weight] => -3 [#title] => File [#access] => 1 [#label_display] => above [#view_mode] => teaser [#language] => und [#field_name] => field_allegato_file [#field_type] => file [#field_translatable] => 0 [#entity_type] => node [#bundle] => allegato [#object] => stdClass Object ( [vid] => 462310 [uid] => 8831 [title] => 2024RTT02_RISERVATO - Allegato 11 - Verbale 2 - Elenco candidati e convocazione [log] => [status] => 1 [comment] => 0 [promote] => 1 [sticky] => 0 [nid] => 111599 [type] => allegato [language] => it [created] => 1719304975 [changed] => 1720083926 [tnid] => 0 [translate] => 0 [revision_timestamp] => 1720083926 [revision_uid] => 102 [taxonomy_vocabulary_2] => Array ( ) [taxonomy_vocabulary_8] => Array ( ) [body] => Array ( ) [field_titolo_frontend_all] => Array ( [und] => Array ( [0] => Array ( [value] => Verbale 2 - Elenco candidati e convocazione [format] => [safe_value] => Verbale 2 - Elenco candidati e convocazione ) ) ) [field_allegato_file] => Array ( [und] => Array ( [0] => Array ( [fid] => 131620 [uid] => 32 [filename] => Verbale 2 ELENCO E CONVOCAZIONE - 2024RTT02 RISERVATO ALL.11 IUS13.pdf.pdf [uri] => public://2024/Verbale 2 ELENCO E CONVOCAZIONE - 2024RTT02 RISERVATO ALL.11 IUS13.pdf.pdf [filemime] => application/pdf [filesize] => 310710 [status] => 1 [timestamp] => 1719304966 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2614 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) ) [name] => carriere.docenti [picture] => 0 [data] => b:0; [num_revisions] => 1 [current_revision_id] => 462310 [is_current] => 1 [is_pending] => [revision_moderation] => [entity_view_prepared] => 1 ) [#items] => Array ( [0] => Array ( [fid] => 131620 [uid] => 32 [filename] => Verbale 2 ELENCO E CONVOCAZIONE - 2024RTT02 RISERVATO ALL.11 IUS13.pdf.pdf [uri] => public://2024/Verbale 2 ELENCO E CONVOCAZIONE - 2024RTT02 RISERVATO ALL.11 IUS13.pdf.pdf [filemime] => application/pdf [filesize] => 310710 [status] => 1 [timestamp] => 1719304966 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2614 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) [#formatter] => file_default [0] => Array ( [#theme] => file_link [#file] => stdClass Object ( [fid] => 131620 [uid] => 32 [filename] => Verbale 2 ELENCO E CONVOCAZIONE - 2024RTT02 RISERVATO ALL.11 IUS13.pdf.pdf [uri] => public://2024/Verbale 2 ELENCO E CONVOCAZIONE - 2024RTT02 RISERVATO ALL.11 IUS13.pdf.pdf [filemime] => application/pdf [filesize] => 310710 [status] => 1 [timestamp] => 1719304966 [type] => document [field_folder] => Array ( [und] => Array ( [0] => Array ( [tid] => 2614 ) ) ) [metadata] => Array ( ) [display] => 1 [description] => ) ) ) [links] => Array ( [#theme] => links__node [#pre_render] => Array ( [0] => drupal_pre_render_links ) [#attributes] => Array ( [class] => Array ( [0] => links [1] => inline ) ) [node] => Array ( [#theme] => links__node__node [#links] => Array ( [node-readmore] => Array ( [title] => Read more about 2024RTT02_RISERVATO - Allegato 11 - Verbale 2 - Elenco candidati e convocazione [href] => node/111599 [html] => 1 [attributes] => Array ( [rel] => tag [title] => 2024RTT02_RISERVATO - Allegato 11 - Verbale 2 - Elenco candidati e convocazione ) ) ) [#attributes] => Array ( [class] => Array ( [0] => links [1] => inline ) ) ) ) )

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